A vein-viewing application enabled detecting abdominal wall varices related to the presence of non-treated gastroesophageal varices: a cross-sectional study

Table 1 Characteristics of patients

Patients	n = 100
Sex (male:female)	71:29
Age (years)	70.5 (20–87)
Etiology of liver disease
HBV infection	19
HCV infection	21
Alcoholism	33
PBC	3
AIH	4
Others^†	20
The status of the underlying liver disease
Chronic hepatitis	26
Cirrhosis	74
Child–Pugh classification
A:B:C	37:25:12
Esophageal varices^‡	57
Location (Li:Lm:Ls)	10:34:13
Form (F1:F2:F3)	26:29:2
Color (Cw:Cb)	56:1
RC0:RC1:RC2:RC3	30:18:6:1
Gastric varices^†	24
Location (Lg-c:Lg-f:Lg-cf)	13:10:1
Form (F1:F2:F3)	14:7:3
Color (Cw:Cb)	14:10
RC0:RC1:RC2:RC3	24:0:0:0
Sarin’s classification
GOV1:GOV2:GIV1:GIV2	12:8:4:0
Past treatment of GOV	30
EVL:EIS:B-RTO:Hassab	21:6:2:1
Past rupture history	12
Portal hypertensive gastropathy	27
Encephalopathy	4
Ascites	32
Splenomegaly	53
Portosystemic shunt	15
SR:GR:IMC	8:6:1

AIH autoimmune hepatitis, Cw white varices, Cb blue varices, F1 straight, small-caliber varices, F2 moderately enlarged, beady varices, F3 markedly enlarged, nodular or tumor-shaped varices, GOV gastroesophageal varices, HBV hepatitis B virus, HCV hepatitis C virus, Ls locus superior, Lm locus medialis, Li locus inferior, Lg-c adjacent to the cardiac orifice, Lg-cf extension from the cardiac orifice to the fornix, PBC primary biliary cholangitis, RC red color sign, SR splenorenal shunt, GR gastrorenal shunt, IMC inferior mesenteric caval shunt, Data are expressed as median (range)
^†Including Budd–Chiari syndrome and cryptogenic, ^‡including treated patients

ISSN: 1471-2342